Project 3 - Walter Rosenthal
   
Date of Birth: 11 October 1954
Nationality: German
   
EDUCATION:  
1974 - 1980
Medical student, University of Gießen; Royal Free Hospital, London
1977 - 1982
Thesis, Rudolf-Buchheim-Institut, University of Gießen
1983-1990
Research on G-proteins: Universities of Heidelberg and Berlin; Baylor College of Medicine, Houston; 1990 habilitation
 
 
PROFESSIONAL EXPERIENCE:
 
1991-1993
Heisenberg fellow, visiting professor, Baylor College of Medicine, Houston
1993-1996
C4-professorship, chair, Rudolf-Buchheim-Institut, University of Gießen
1987-1988
Recipient of a “Habilitandenstipendium” from the “Deutsche Forschungsgemeinschaft” (Competitive German Grant), Max-Planck-Institut für Entwicklungsbiologie, Tübingen
1988-1993
Research group leader at the Zentrum für Molekulare Neurobiologie, Universitätskrankenhaus Eppendorf, Hamburg
since 1996
Director of the FMP
1998-2003
SC4-professorship, Institut für Pharmakologie (Dahlem), Free University of Berlin
since 2003
C4-professorship, chair, Institut für Pharmakologie, Charité (Dahlem)
 since 2005
Spokesman, Life Science Section of the Leibniz Association
 since 2005
Spokesman, Board of Directors, Forschungsverbund Berlin e.V. (8 Leibniz institutes, 1100 employees)
HONORS/AWARDS
 
1980
Scholarship of the German Academic Exchange Service (DAAD)
1983
Scholarship of the Boehringer Ingelheim Fonds
1991-1993
Heisenberg scholarship of the German Research Foundation (DFG)
RESEARCH INTERESTS:
Major research areas:
Intracellular protein trafficking, scaffolding proteins, G-protein-coupled receptors, cAMP/PKA-triggered pathways, aquaporins, molecular defects underlying human disease, pharmacological interference
Highlights:
Regulation of ion channels by G-proteins; assignment of functions to subtypes of G-Protein subunits; molecular cloning of the human V2 (ADH) receptor; first description of disease-causing mutations in a G protein-coupled hormone receptor; transport and processing of G-protein-coupled receptors; molecular basis of cAMP-triggered exocytosis (aquaporin 2 shuttle); disruptors of protein-protein interactions
COOPERATIVE RESEARCH ACTIVITIES:
Board member, principal investigator: SFB 366 “Cellular Signal Recognition and Transduction”
Board member, principal investigator: SFB 449 “Structure and Function of Membrane Proteins”
Member DFG Research Group 341 “Mechanisms and Pathomechanisms of Vascular Regulation”
Member Graduate School “Signal recognition and translation” (GRK 276)
Principal investigator, EU QLK3-CT-2002-02149 “Anchored cAMP signalling”


TOP PUBLICATIONS:

 
1.
Kaufmann JE, Oksche A, Wollheim CB, Günther G, Rosenthal W, Vischer UM (2000) Vasopressin-induced von Willebrand factor secretion from endothelial cells involves V2 receptors and cAMP. J Clin Invest 106: 107-116
2.
Marr N, Bichet DG, Lonergan M, Arthus MF, Jeck N, Seyberth H, Rosenthal W, van Os CH, Oksche A, Deen PMT (2002) Heteroligomerisation of an aquaporin-2 mutant with wild-type aquaporin and their misrouting to late endosomes/lysosomes explains dominant nephrogenic diabetes insipidus. Hum Mol Genet 11: 779-789
3.
Lorenz D, Krylov A, Hahm D, Hagen V, Rosenthal W, Pohl P, Maric K (2003) Cyclic AMP is sufficient for triggering the exocytic recruitment of aquaporin-2 in renal epithelial cells. EMBO Report 4: 88-93
4.
Henn V, Edemir B, Stefan E, Wiesner B, Lorenz D, Theilig F, Schmitt R, Vossebein L, Tamma G, Beyermann M, Krause E, Herberg FW, Valenti G, Bachmann S, Rosenthal W, Klussmann E (2004) Identification of a novel A-kinase anchoring protein 18 isoform and evidence for its role in the vasopressin-induced aquaporin-2 shuttle in renal principal cells. J Biol Chem 279: 26654-26665
5.
Hermosilla R, Oueslati M, Donalies U, Schönenberger E, Krause E, Oksche A, Rosenthal W, Schülein R (2004) Disease-causing V2 vasopressin receptors are retained in different compartments of the early secretory pathway. Traffic 5: 993-1005
6.
Giebing G, Tölle M, Jürgensen J, Eichhorst J, Furkert J, Beyermann M, Neuschäfer-Rube F, Rosenthal W, Zidek W, van der Giet M, Oksche A (2005) Arrestin-independent internalization and recycling of the urotensin receptor contribute to long-lasting urotensin II-mediated vasoconstriction. Circ Res 97: 707-715

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