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| Ali
Poyan Mehr |
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Home
Institution
Freie Universität Berlin
Host
Institution(s)
Yale University School of Medicine, New Haven
Research Mentor: John Forrest, M. D.
E-Mail:
poyanmehr@web.de
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Research
Topic
see Abstract |
Personal
Reactions to the U.S. Experience
Spending one year in U.S. to do some research is an experience you should
not miss. But, fortunately, experience in science (and maybe barbeque sauce
and sneakers) are not the only things you will carry home. Of course, because
of my citizenship (Iran), I belonged to "the axis of evil". This circumstance
made it a "little bit" difficult for me the get the visa. But as soon as
I had it, and entered the US, I didn't experience any of the animosities
I expected. Never! Most of the US Americans are extremely polite, which
is one of many cultural characteristics I appreciated. And there are also
many other things we can learn from. Just go and find out! |
Greatest
Difficulties Encountered
Taking a look through what other student have written in previous years
regarding difficulties, there is nothing I can add. And I don't want to
repeat them just because I think there are not really any serious difficulties
you will face. Hmm
the greatest difficulty I encountered was finding some
souvenirs before going back to Germany. This should give you an impression
how easy life can be. |
Most
humorous incident
During my stay at Mount Desert Island Bio labs in summer, some friends and
I bought half a dozen crabs and lobsters to make a big dinner. Of course
they were fresh and alive when we left them in the car, inside a plastic
bag, just for a short moment. Well, you can guess what happened after we
came back. The bags were open, the car flooded with sea water, and we had
to find our dinner under the seats and everywhere else they could hide.
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Helpful
Hints for Future Students
- My visa
application was rejected three times. Don't give up and try hard to
get what you want. Of course you will need the help of your mentor for
this.
- Get in
touch with other student and international scholars. Feel free to contact
me regarding any kind of question you have.
- Be careful
riding a bike, but do it! You can save a lot of money!
- Put some
money into your saving account. It will help you at the beginning.
- Get your
social security number as soon as possible.
- Be open
minded
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| Abstract
on Research Topic |
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Mercury
sensitivity of shark CFTR is mediated through cysteine residues located
in the first transmembrane domain
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Authors:
Ali Poyan Mehr and John Forrest, M.D. |
Institution:
Department of Nephrology, School of Medicine, Yale University |
Purpose:
There are profound differences between mercury inhibition of shark CFTR
vs. human CFTR. Cysteine residues (Cys) are the known biding sites for mercury,
thus mercury can exhibit its toxic effects on a countless number of proteins.
Correlating specific Cys in certain protein domains with mercury toxicity
can contribute to the understanding of the function and importance of this
domain. Specifically in channel proteins those findings may promote a clearer
vision of the three dimensional structure and the arrangement of the pore
region of a protein. Both shark and human CFTR share 12 cysteine residues,
including three, but in addition the shark homolog has 2 unique in the first
transmembrane domain (TMD1) and 2 in the regulatory domain (RD). One or
more of these unique Cys residues are most likely mediating the extreme
higher sensitivity of sCFTR to mercury versus hCFTR. In order to approach
the question, whether the Cys in TMD1 or those in RD are mediating this
effect, we constructed chimeric proteins where we replaced the human TMD1
and RD with the shark analogues respectively. Using Xenopus laevis oocytes
and TEVC we could explore which part of the shark CFTR carries the mercury
sensitivity in to the human CFTR. |
Materials
and Methods:
Chimeric proteins were constructed using a combination of PCR and restriction
enzyme digestion. The TMD1 of shark was amplified using sense primer with
an overlapping sequence homolog to the N-terminal end of hCFTR and an antisense
primer with an introduced restriction site for BsiW I. The N-terminal end
of hCFTR was amplified in a separate step. Both fragments were then combined
together in a third reaction. The product was finally combined with the
rest of hCFTR, and cloned in to pBlueskript KS(-). Analog to TMD1 the RD
was amplified with primer containing BstZ17 I and Hpa I restriction sites
and cloned in to hCFTR lacking its own RD. The TMD1 chimeras as well as
the wiled types were injected into Xenopus laevis oocytes and TEVC was performed
after two to three days of incubation. After a short recovery time in ND96,
chloride conductance was activated by adding 10µM forscolin and 1mM isobutylmethylxanthine
(IBMX). Once the oocytes reached a plateau (usually after 25 to 35 minutes),
HgCl was then added to the solution in a concentration of 1µM. |
Results:
From the RD chimera there are not enough data available yet for a statistical
analysis, but the degree of inhibition is similar to hCFTR. TMD1 shows the
same degree of inhibition like wild type sCFTR (89.20% and 94.58 respectively,
p<0.0001). |
Conclusion:
Cys residues located in the first transmembrane domain of shark CFTR are
likely to be located within the pore forming unit, which is not well described
yet. Site specific mutagenesis of those residues will help to find the arrangement
of the pore region in more detailed and help to understand the structure-function
relationship of CFTR and channel proteins in general. |